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Cephalalgia Award Lecture winner announced

May 10th, 2011

The winner of the Cephalalgia Award Lecture, which is awarded to the best paper submitted and/or published in the journal Cephalalgia between January 1, 2010 and February 28, 2011, was announced today. The award winning paper is A new trigemino-nociceptive stimulation model for event-related fMRI by A Stankewitz, HL Voit, U Bingel, C Peschke and A May Cephalalgia 2010; 30: 475.

The authors will receive €10,000 and will be presenting their paper at the award lecture which will be given at the 15th Congress of the International Headache Society in Berlin on Saturday, June 25, 2011 from 18:00-18:30 in Hall A in the congress center. From more information please visit: http://www2.kenes.com/ihc2011/Pages/Home.aspx

"We are overwhelmed and thank all the jurors" said Professor Arne May on behalf of the winning author team. "It is a great honor to be nominated for such a prestigious award and means very much for us. It is also a great help in visualizing and stimulating future headache-research in Germany which in itself is fundamentally important and highly valuable."

"The International Headache Society and Cephalalgia's editorial board is very proud of the high quality clinical and basic science papers published in the journal," said Professor David Dodick, Editor of Cephalalgia. "Nowhere is this quality and cutting-edge innovation more evident than in the paper awarded the prestigious Cephalalgia Award for 2010-2011. The study by Stankewitz and colleagues was selected by the editorial board as the best paper based on its innovation, scientific rigor, and the potential to advance the field of headache science. The trigeminal-noiceptive fMRI paradigm constructed by the authors is non-invasive, provides high spatial resolution, and may be used to investigate and characterize the specific anatomical pathways and functional brain abnormalities involved in migraine and other primary headache and facial pain disorders, as well as provide anatomical mapping of pharmacologically induced modulation of these pathways."

Provided by SAGE Publications

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